Gurpreet Kaur’s journey in this world ....
Gurpreet Kaur was a Musician. She was a singer and a composer of music. Her interest was composing and singing Gurbani Shabads in Indian Classical style. She sang Shabads in All the Raags mentioned in Sri Guru Granth Sahib Ji. She also taught Gurmat Sangeet at Gurmat Gian Missionary College, Jawadi, Ludhiana.
Elder child to Pushpinder Kaur and Dr. Brig. Harminder Singh, was born in Amritsar on 13th Jan 1962. She attended various convent schools as a child because her father would get frequent Army postings as a dental surgeon. She graduated with Music Honors from Govt. College for Women, Chandigarh.
Music was her hobby and she composed and sang Raag based Gurbani Shabads. Doing Kirtan was part of growing up nurtured by her parents. She learned music from her father Dr. Brigadier Harminder Singh who was a dental surgeon in Indian Army and a very good singer himself. Gurpreet’s Bhua (father’s sister), Ajit Kaur retied as a Head of Department of Music from Govt. College for Women Ludhiana, and was a renounced Punjabi singer of her time. Gurpreet Kaur also learned nuances of Indian Classical Music from Pandita Sharma.
She was a mother of three children, and a grandmother. Her daughter Keerat Kaur is a Computer Engineer. Her two sons Gurkeerat Singh and Jaskeerat Singh are doctors in USA. Her daughter Keerat Kaur too was part of her group ~ Gurmat Gian Group.
Gurpreet Kaur left this world at the age of 54yrs on 12th Sept 2016 in Baltimore USA.
She had recorded around 25 cds of Gurbani Keertan.
'Raag Ratan' Album (6 CDs) is a Compilation of Shabads in All the 31 Sudh Raags of Sri Guru Granth Sahib Ji.
'Gauri Sagar' Album (3 CDs) is a Compilation of All forms of Raag Gauri in Sri Guru Granth Sahib Ji.
'Nanak Ki Malhaar' ~ ((3 CDs) is an album of Raag Malhar Shabads in various forms of Malhar.
'Gur Parsaad Basant Bana' ~ (3 CDs) is an album of Shabads in Raag Basant sung in various forms of Raag Basant.
Har Ki Vadeyai
Sarni Aayea
Sewa
Priya Kee Preet Piyaree
Mohan Ghar Aavho Karo Jodariya
Mo Kao Taar Le Raama Taar Le
Tere Kavan Kavan Gun Keh Keh Gawan
Mera Baid Guru Govinda
Saajanrraa Mera Saajanrraa
Obesity is one of the most significant health problems in the developing and developed countries. An overweight adolescent has a 70 percent chance of becoming an obese adult, and obesity can lead to a higher risk of life threatening health problems like –
High blood pressure
Premature heart attacks
Diabetes
Physical risks
Emotional ramifications because of peer ridicule and lower self-esteem levels.
The additional weight can result in reduced endurance, leg pains from stressed joints and often make simple activities more taxing.
All this leads to “The Vicious Cycle of Childhood Obesity”.
It is believed that children are becoming heavier due to –
decrease in physical activity (modern transportation, television, computers and video games)
Increasing availability of higher calorie foods such as fast foods, higher-fat convenience foods and snacks.
Management of Childhood Obesity
Dietary intervention.
An exercise program.
Behaviour counselling with family participation.
There is a rare genetic disorder that is related to childhood obesity. It is named Prader-Willi Syndrome, a rare genetic disorder that affects about one of every 15,000 people. Prader-Willi Syndrome has been recognized as the most common genetic cause of potentially life-threatening childhood obesity.It results from an abnormality on the 15 chromosome.The children have extraordinarily low metabolism and an insatiable hunger and insatiable urge to eat, which is beyond the control of the patient.
Other features of this condition are:
Low muscle tone and short stature
Small hands and feet
Poor motor skills
Reduced bone mineral density.
Delayed or incomplete sexual development
Mild to moderate mental retardation
Distinctive facial features (including a narrow face, small mouth, and full cheeks)
Obesity What is more Important – Your Weight or Your Shape ?
The ultimate risk of diseases in relation of obesity is not the weight but the shape of the weight your body carries.
Now many studies have established the fact that if you have a pear shape – namely carrying most of your fat in your butt and your thighs – you are better off than an apple shape where your waist measurement is beyond your hip measurement.
Here is an eight-year follow-up to the nurse’s health study which began with almost 45,000 women. A definite pattern emerged which tells you that it’s not how much you weigh, but where that weight is that can be critical for heart disease. For woman, risk for dying from heart disease is much greater than the risk of dying from breast cancer and yet, the latter seems to get a lot more attention and fear.To find out what the risks are this simple test would help.First, let’s start with waist-hip ratio.To measure your waist-hip ratio, measure your waist at the level of your belly button and your hips at the largest circumference with a tape measure.On division of waist and hip measures we get the waist -hip ratio which should be somewhere between 0.7 to 1.0.If it’s above one that implies your waist is larger than your hips. That’s not good.
Now let’s go for the body mass index. The body mass index, or BMI, is the ratio of your weight in kilograms divided by your height in meters squared.
It is now found that even if your BMI is higher than desirable, it doesn’t seem to make as much difference as your waist-hip ratio.BMI’s of less than 25 has been considered desirable, over 25 is considered to be undesirable.
It is also to be understood that the risk for women under the age of 60 is more pronounced than women over the age of 60. The risk factors that we are concerned here are mainly for coronary heart disease which includes non fatal heart attacks as well as fatal heart attacks.
If your waist-hip ratio is 0.72 or below, you are the standard, and don’t at least have to worry about heart attacks based on your body shape.If your waist-hip ratio is 0.72 to .76 and you are under 60, your risk is 1.7 times higher than the standard or 70 percent higher.If your ratio is 0.76 to 0.80, your risk is two and a half times higher.If your ratio is 0.80 to .88, your risk approaches three times higher.If your waist-hip ratio is over 0.88, your risk is four and a half times higher.If you are older than 60, the top category of risk is 1.9 times higher.So you can see that the effects of this are diminished in women over the age of 60, implying that extra abdominal fat is less risky for older woman. Now here is the major point of this study. With minor exceptions, it didn’t matter whether your body mass index was under 25 or over 25. In other words, an overweight woman, if she has more of an hour-glass figure, seems to be protected. In addition, you can look at the absolute waist circumference measurements.
At 71.1 centimetres, your waist is fine.At a waist circumference of 81.3 to 86.4 centimetres, your risk is a couple of times higher.If your ratio is over 96 centimetres, and you are under 60, your risk is up four times. If you are over 60, your risk is only up two times.
It can be easily concluded that an overweight woman, if she has more of
an hour-glass figure, seems to be protected.
A waist circumference above 100 cm in men and above 90 cm in women is associated with increased levels of triglyceride and reduced levels of HDL cholesterol.
So, for those who are in a high risk category it’s important to take preventive measures concerning exercise and cholesterol and take care of oneself as best as possible.
Obesity is an excess of body fat frequently resulting in a significant impairment of health.
Adipose tissue is a normal constituent of the human body that serves the function of storing energy as fat. The fat may be used by the body in response to various metabolic demands. The excess fat accumulation is associated with increased fat cell size.A very important reason for obesity is the overconsumption of carbohydrates. When carbohydrates enter our body they are rapidly broken down into glucose (a sugar). Glucose raises blood sugar levels causing pancreas to make the hormone insulin. Insulin inhibits fat already stored in our body from being broken down and it also promotes the storage of new fat in the body; and increases cholesterol levels.In many forms of severe childhood-onset obesity, the total number of fat cells is increased.Formerly, obesity was explained by the single adverse behaviour of inappropriate eating in the setting of attractive foods. But now it has been established that obesity has multiple causes and is of different types. Both genetic and environmental factors are likely to be involved in the pathogenesis of obesity. These include excess caloric intake, decreased physical activity, and metabolic and endocrine abnormalities.Genetic determinants can either play a major role in the pathogenesis of obesity or enhance susceptibility to its development.The conditions in which genetics may play a role are Prader-Willi syndrome, Ahlstrom’s syndrome, the Laurence-Moon-Biedl syndrome, Cohen’s syndrome, and Carpenter’s syndrome.Over weight can be estimated by Body mass index (BMI), which is weight/height square, where weight is in kilograms and height is in meters.
Obesity is defined as a body mass index above 30 kg/square meter.
Weight gain may confer increased health risks even if the BMI does not exceed 25. In women a weight gain of more than 5 kg (11 lb) is associated with increased risks of diabetes and heart disease, and in men any weight gain after age 25 appears to carry increased health risks.
More than 30% of Americans are 20% or more overweight, and one third of women and more than one quarter of men are trying to lose weight at any given time.
Jacqueline Noonan, paediatrician and heart specialist in 1963, published a report on a small group of patients with typical facies, congenital heart defect, and some clinical features similar to Turner syndrome, but with normal chromosomes. Its after her that this syndrome has been named.
Noonan syndrome is inherited as an autosomal dominant condition. This means that the Noonan gene is on a non-sex (autosomal) chromosome and is transmitted from parent with a 50% probability to child. Although one dose of the Noonan gene is enough to cause the syndrome. A gene for Noonan syndrome (NS1) has been mapped to chromosome number 12. There are several different Noonan genes.
The frequency of the Noonan syndrome is estimated to be between 1:1,000 and 1:2,500 in the general population
Main features of this syndrome are:
Pulmonary Stenosis
Short stature after birth
Webbing of Neck
Caved in Chest Bones
In boys, Testes that do not descend into the scrotum.
Other possible heart defects are hypertrophic cardiomyopathy (thickened heart muscle), atrial septum defect (hole in the wall separating the anterior heart chambers), ventricular septum defect(hole in the wall separating the main heart chambers), septal hypertrophy or a combination of all these defects.
Other Facial features that may be present in this syndrome are:
Drooping of eyelids.
Downwards slanting eyes with arched eyebrows
Extra skin fold at the inner angle of the eyes.
Increased distance between the eyes.
Broad forehead
Very Blue or blue green eyes
Curly hair
Flat broad root of the nose.
Short broad neck with skin folds
Many pigmented birth marks
Variations occur in the facial features and they change with the age. As the child grows, the face gradually becomes more normal, often more triangular with a high forehead.
Other features of this syndrome
The birth length and weight of children with Noonan syndrome are often normal. The increase in height in both boys and girls is less than normal throughout the whole growing period.
On an average, puberty is delayed by about two years in both sexes.
During the first years of life the children may have great feeding and nutrition problems and frequent respiratory tract infections.
Only a few children with Noonan syndrome are seriously handicapped. Delayed motor development and speech may be seen in about 25% of the children.
Studies have shown that these children’s IQ was within normal limits, though in most cases in the lower range of normal. Mental retardation in approx. 35 %
Many of those with Noonan syndrome have a form of visual disturbance, squinting, near sight, or long sight, which may necessitate use of spectacles. The visual disturbance is usually mild.
A mild type of hearing impairment has been reported.
Children with Noonan syndrome often have dental problems. 1/3 of them have wrongly positioned teeth. The teeth often appear late and in an abnormal order.
Repeated attacks of epilepsy have been described in a few cases
Reduced fertility in men. Women’s fertility is normal.
Nitric oxide is a naturally occurring molecule found in a variety of cell types and organ systems. In the cardiovascular system, NO is an important determinant of basal vascular tone, prevents platelet activation, limits leukocyte adhesion to the endothelium, and regulates myocardial contractility. NO may also play a role in the pathogenesis of common cardiovascular disorders, including hypotension accompanying shock states, essential hypertension, and atherosclerosis.
It was first identified as endothelial-derived relaxing factor in blood vessels and as the mediator of the bactericidal actions of macrophages. NO’s role as a neural messenger may be even more prominent. NO is a likely transmitter of nonadrenergic, noncholinergic neurons. Excess release of NO appears to account for a major portion of neural damage following vascular stroke.
Nitric oxide is a cytotoxic agent of macrophages, a messenger molecule of neurons, and a vasodilator produced by endothelial cells.
Nitric Oxide has been shown to be a mediator of Erectile function.
NO is a critical determinant of basal vascular tone, and a deficiency of NO is associated with hypertension.
Common disorders that promote atherosclerosis, such as hypertension, hyperlipidemia, smoking, and diabetes, are all associated with abnormal endothelial function. Deficiency of bioactive NO is associated with Abnormal Endothelial Function.
A deficiency of NO producing neurons in the gastrointestinal tract is believed to be responsible for certain abnormalities in gastrointestinal motility, such as Hirschsprung’s disease, achalasia, and chronic intestinal pseudo-obstruction.
Drugs like nitroglycerin, isosorbide mononitrate and dinitrate, and nitroprusside, promote vasodilation and platelet inhibition in dysfunctional coronary arteries by being metabolized to NO.
NO can be useful in the treatment of persistent pulmonary hypertension of the newborn, the pulmonary vasoconstriction that accompanies congenital diaphragmatic hernia, primary pulmonary hypertension, and adult respiratory distress syndrome.
attacks based on your body shape.If your waist-hip ratio is 0.72 to .76 and you are under 60, your risk is 1.7 times higher than the standard or 70 percent higher.If your ratio is 0.76 to 0.80, your risk is two and a half times higher.If your ratio is 0.80 to .88, your risk approaches three times higher.If your waist-hip ratio is over 0.88, your risk is four and a half times higher.If you are older than 60, the top category of risk is 1.9 times higher.So you can see that the effects of this are diminished in women over the age of 60, implying that extra abdominal fat is less risky for older woman. Now here is the major point of this study. With minor exceptions, it didn’t matter whether your body mass index was under 25 or over 25. In other words, an overweight woman, if she has more of an hour-glass figure, seems to be protected. In addition, you can look at the absolute waist circumference measurements.
Formerly, obesity was explained by the single adverse behaviour of inappropriate eating in the setting of attractive foods. But now it has been established that obesity has multiple causes and is of different types. Both genetic and environmental factors are likely to be involved in the pathogenesis of obesity. These include excess caloric intake, decreased physical activity, and metabolic and endocrine abnormalities.Genetic determinants can either play a major role in the pathogenesis of obesity or enhance susceptibility to its development.The conditions in which genetics may play a role are 
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